BẤT THƯỜNG TÍN HIỆU TÚI TINH TRÊN CỘNG HƯỞNG TỪ Ở BỆNH NHÂN CÓ RỐI LOẠN XUẤT TINH

Abstract

Purposes: To eValuate the abnormal signal intensity of seminal Vesicles in the group of patients with ejaculation disorder. Material and methods: A descriptiVe study on abnormal signal intensity of seminal Vesicles in 71 patients who were in male examination at Hanoi Medical UniVersity Hospital due to ejaculation disorders (infertility, hematospermia.) and underwent MRI of the seminal Vesicles from 8/2020 to 10/2022. Result: mean age was 34.8 ±

12.5 in which 42 patients had hematospermia, 21 patients came to the clinic because of infertility, 3 patients had uricle cysts, 2 patients with bilateral seminal Vesicle cysts, 2 patients with bilateral seminal Vesicle dilatation, 1 patient with ejaculatory duct cysts. Among 42 patients with hematospermia, there were 12 patients with seminal Vesicles that were isointense on T1W, hyperintense on T2W (accounting for 29%), there were 8 patients with hyperintense seminal Vesicles on T1W and T2W (accounting for 19%), there were 22 patients with seminal Vesicles with increased signal on T1W, decreased signal on T2W (accounting for 52%), There are 7 patients with seminal Vesicle stones (accounting for 17%) presented by signal Void on T1W and T2W. There are 6 patients (accounting for 14%) with thickening of the seminal Vesicle wall corresponding to the inflammation of seminal Vesicles. Of the total of 21 patients with congenital abnormality of the seminal Vesicles, there were 4 patients with bilateral seminal Vesicle aplasia, accounting for 19%, there were 3 patients with unilateral seminal Vesicle aplasia, accounting for 14.2%, and 6 patients with unilateral hypoplasia of seminal Vesicles and 6 patients with normal seminal Vesicles, accounting for 28.6%, respectiVely. There was 1 patient with bilateral seminal Vesicle hypoplasia, 1 patient with seminal Vesicle hypoplasia on one side with aplasia of the other seminal Vesicles, accounting for 4.8%, respectiVely. All hypoplastic seminal Vesicles (either unilateral or bilateral) are hyperintensity on T1WFS. There were 8 patients with ejaculation disorder due to other causes, including 2 patients with seminal Vesicle cyst, 2 patients with dilated seminal Vesicles, 3 patients with utricule cyst and 1 patient with ejaculatory duct cyst. These cysts present with a hypointense structure on T1-weighted images and hyperintensity on T2- weighted images. Conclusion: Most of the patients with ejaculatory disorder had abnormal hyperintensity on T1W of seminal Vesicles (due to hematospermia or sperm obstacle). The detection of signal abnormalities of the seminal Vesicles was important in detecting the cause of ejaculation disorders.