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CLINICAL AND IMAGING CHARACTERISTICS OF DEEP CEREBRAL INFARCTION OF MIDDLE CEREBRAL ARTERY
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Abstract
Objectives: Determine suggesting the possible mechanism and etiology of subgroups of deep cerebral infarction. Subjects and methodology: this is a retrospective comparative study, based on patients with deep cerebral infarction of the middle cerebral after admitted to University Medical Center during the study period. Results: Our study enrolled 157 patients with deep cerebral infarction in the middle cerebral artery, which showed some results as follows: the male/female ratio is 1.7; mean age is around 60. Hypertension and dyslipidemia are common vascular risk factors (93.6% and 74.5%, respectively). Atrial fibrillation is only seen in the striatocapsular infarction affected both distal and proximal region, with a rate of 46.7%. Indicators of small vessel disease (white matter hyperintensities with Fazekas ≥ 2, microbleeds, silent brain infarction) are more common in the distal single small subcortical infarction (64.2%, 23.9%, and 26.9%, respectively). Indicators of atherosclerosis (parent artery disease and atherosclerosis of other cerebral arteries) are common in the proximal single small subcortical infarction (both at 42.3%) and striatocapsular infartion. Conclusion: Distal single small subcortical infarction suggests etiology of small vessel disease, striatocapsular infarction affected both distal and proximal region suggest cardioembloism, and proximal single small subcortical infarction and striatocapsular infarction suggest parent artery athreoscelrosis.
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Keywords
Deep cerebral infarction, middle cerebral artery, striatocapsular infarction, single small subcortical infarction
References
2. Nguyễn Văn Thành, Nguyễn Thị Điểm, Huỳnh Thị Phương Minh (2012). Đặc điểm nhồi máu não lỗ khuyết. Y học TPHCM, 16(4): 624-630.
3. Del Bene A., Palumbo V., Lamassa M., et al. (2012). Progressive Lacunar Stroke: Review of Mechanisms, Prognostic Features, and Putative Treatments. International Journal of Stroke, 7(4):321-329.
4. Halkes PH, Kappelle LJ, van Gijn J., et al. (2006). Large subcortical infarcts: clinical features, risk factors, and long-term prognosis compared with cortical and small deep infarcts. Stroke, 37(7):1828-32.
5. Bae YJ, Choi BS, Jung C., et al. (2017). Differentiation of Deep Subcortical Infarction Using High-Resolution Vessel Wall MR Imaging of Middle Cerebral Artery. Korean J Radiol, 18(6):964-972.
6. Lee KB, Oh HG, Roh H., et al. (2008). Can we discriminate stroke mechanisms by analyzing the infarct patterns in the striatocapsular region? Eur Neurol, 60(2):79-84.
7. Djulejić V., Marinković S., Maliković A., et al. (2012). Morphometric analysis, region of supply and microanatomy of the lenticulostriate arteries and their clinical significance. J Clin Neurosci, 19(10):1416-21.
8. Sun LL, Li ZH, Tang WX, et al. (2018). High resolution magnetic resonance imaging in pathogenesis diagnosis of single lenticulostriate infarction with nonstenotic middle cerebral artery, a retrospective study. BMC Neurol, 18(1):51.
9. Wen L., Feng J, Zheng D. (2013). Heterogeneity of single small subcortical infarction can be reflected in lesion location. Neurol Sci, 34(7):1109-16.
10. Nah HW, Kang DW, Kwon SU, et al. (2010). Diversity of single small subcortical infarctions according to infarct location and parent artery disease: analysis of indicators for small vessel disease and atherosclerosis. Stroke, 41(12):2822-7.
11. Kim BJ, Yoon Y., Lee DH, et al. (2015). The shape of middle cerebral artery and plaque location: high-resolution MRI finding. Int J Stroke, 10(6):856-60.